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Ozone Hazards.

Authors
Anonymous
Source
NIOSH 1977 Oct:47-50
NIOSHTIC No.
00075412
Abstract
This information profile on ozone (10028156) lists its chemical structure, Chemical Abstracts Service number, Registry of Toxic Effects of Chemical Substances number, production figures and economic trends in production, uses, number of companies producing and using ozone, and the biological effects of exposure to ozone. The lungs are the primary site of ozone induced tissue damage with pulmonary congestion, edema, and hemorrhage resulting. Subchronic exposures result in significant changes in lung function. Ozone may cause a defect in oxygen dissociation from oxyhemoglobin. Ozone is radiomimetic and may cause decreased life span in chronically exposed animals. Acute, subchronic, and chronic effects of exposure are discussed. Occupational exposure to humans at 2ppm can cause pulmonary congestion. The outcome of intelligence tests was not affected by ozone exposures of 0.3ppm for 70 minutes. Visual acuity was reduced by ozone at 0.2ppm. The current threshold limit value for ozone is 0.1ppm which may cause premature aging in some individuals. (Contract No. 210-77-0120)
Keywords
NIOSH-Contract; Contract-210-77-0120; Blood-disorders; Oxyhemoglobin-dissociation; Anoxia; Respiratory-system-disorders; Vision-disorders; Personality-traits; Toxic-gases; Psychological-factors; Respiratory-functions;
CAS No.
10028-15-6;
Publication Date
19771001
Funding Type
Contract;
Fiscal Year
1978
NTIS Accession No.
NTIS Price
Identifying No.
Contract-210-77-0120
Priority Area
Asthma and Chronic Obstructive Pulmonary Disease; Disease and Injury; Respiratory-system-disorders;
Source Name
Information Profiles on Potential Occupational Hazards, NIOSH Contract No. 210-77-0120, October 1977
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